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Addition of LDL to primary trophoblast cultures drastically suppresses de novo cholesterol synthesis, stimulates progesterone production and inhibits cholesteryl ester-forming ACAT (Winkel et al., 1980a,b, 1981)
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According to previous studies, sepsis is the main cause of ARDS, accounting for 31% of the etiology of ARDS, and ARDS is a serious and destructive complication of sepsis ( 2.6 Genetic variability When studying the genetic characteristics of ARDS, it is arduous to obtain a family pedigree of ARDS patients, because the disease is often secondary to other serious systemic diseases and most cases are sporadic (71)
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