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The cell has several nutrients sensors responsible for maintaining cell homeostasis with the extracellular environment, such as the mammalian/mechanistic target of rapamycin complex 1 (mTORC1) that drives ATP-consuming cellular processes (anabolic) necessaries for cellular proliferation and growth ( In this review we explain the participation of mTORC1 in the regulation of mitochondrial ATP producing capacity and we discuss how this process affects tumor cells
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It reduces IL-1-induced nitric oxide synthesis in human chondrocytes and decreases the production of prostaglandin E2, nitric oxide, and TNF-α/IL-1 in macrophages stimulated with lipopolysaccharides (LPSs)
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