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[10] It is worth being exact about what that source says, because the naive reading goes too far in the other direction: the recommendation is to adjust the parenteral copper dose according to serum concentrations and the clinical picture, explicitly not to stop it, since discontinuing copper in cholestatic patients risks producing copper deficiency instead
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It influences several overlapping repair pathways simultaneously: it may promote new blood vessel growth (VEGF), modulate the nitric oxide system, support collagen synthesis, and protect gut mucosal tissue
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