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In vitro, CD36, FATP, CPT1a, and ACOX1 were significantly downregulated in HK-2 cells treated with COM, recovered after PRMT1 knockdown, and further decreased after PRMT1 overexpression (Fig
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Memory and cognitive function have been demonstrated to be negatively impacted by its administration’s transient blockade of cholinergic muscarinic receptors [180]
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